Oculomotor apraxia

Oculomotor apraxia
Other namesCogan ocular motor apraxia or saccadic initiation failure
SpecialtyOphthalmology

Oculomotor apraxia (OMA) is the absence or defect of controlled, voluntary, and purposeful eye movement.[1] It was first described in 1952 by the American ophthalmologist David Glendenning Cogan.[2] People with this condition have difficulty moving their eyes horizontally and moving them quickly. The main difficulty is in saccade initiation, but there is also impaired cancellation of the vestibulo-ocular reflex. Patients have to turn their head in order to compensate for the lack of eye movement initiation in order to follow an object or see objects in their peripheral vision, but they often exceed their target. There is controversy regarding whether OMA should be considered an apraxia, since apraxia is the inability to perform a learned or skilled motor action to command, and saccade initiation is neither a learned nor a skilled action.[3]

  1. ^ Tada, M.; Yokoseki, A.; Sato, T.; Makifuchi, T.; Onodera, O. (2010). "Early-Onset Ataxia with Ocular Motor Apraxia and Hypoalbuminemia/Ataxia with Oculomotor Apraxia". Diseases of DNA Repair. Advances in Experimental Medicine and Biology. Vol. 685. pp. 21–33. doi:10.1007/978-1-4419-6448-9_3. ISBN 978-1-4419-6447-2. PMID 20687492.
  2. ^ Le Ber, I.; Brice, A.; Dürr, A. (2005). "New autosomal recessive cerebellar ataxias with oculomotor apraxia". Current Neurology and Neuroscience Reports. 5 (5): 411–417. doi:10.1007/s11910-005-0066-4. PMID 16131425. S2CID 29587221.
  3. ^ Galvaez-Jimenez N, Tuite P, Bhatia K, editors. Uncommon Causes of Movement Disorders. New York: Cambridge University Press; 2011:38-40.
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