Intraoral dental sinus

An intraoral dental sinus is an abnormal channel that forms between a periapical infection and the oral cavity, allowing pus to drain into the mouth. It is a common consequence of chronic odontogenic infections, typically resulting from untreated dental caries, pulpal necrosis, or failed endodontic treatment.[1] The condition often presents as a small, erythematous nodule or an opening on the gingiva or alveolar mucosa, which may intermittently discharge purulent material.[2] While patients may experience discomfort during the initial infection phase, pain often subsides once the sinus tract establishes drainage, leading to delayed diagnosis and persistent low-grade infection.[3]

The etiology of intraoral dental sinuses is primarily linked to periapical abscesses, which develop when bacterial infections from the root canal system extend into periapical tissues.[4] The path of sinus tract formation is influenced by anatomical factors such as bone density and muscle attachments, determining whether the infection drains intraorally or extraorally.[5] If left untreated, the infection may progress to more severe complications, including osteomyelitis, cellulitis, or deep fascial space infections.[6]

Correct diagnosis is essential, as intraoral dental sinuses can be misdiagnosed as periodontal abscesses or mucosal lesions of non-odontogenic origin.[7] Clinicians often use radiographic imaging, such as periapical radiographs or cone-beam computed tomography (CBCT), along with gutta-percha tracing to determine the source of infection.[8] Management involves addressing the underlying cause through root canal treatment or tooth extraction, ensuring complete resolution of the infection and closure of the sinus tract.[9]

This review discusses the pathophysiology, clinical presentation, diagnostic approaches, and management strategies for intraoral dental sinuses, emphasizing their significance in dental practice and the importance of timely intervention.

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  2. ^ Gadre, Pallavi (2025-02-27). "Assessment of Intraoral Imaging Exposure Rates Among Dental Students: A Cross-Sectional Study on Radiographic Errors and Retake Factors". International Journal of Science and Research. 14 (2): 823–826. doi:10.21275/sr25211214805. ISSN 2319-7064.
  3. ^ Rôças, I. N.; Siqueira, J. F.; Andrade, A. F. B.; Uzeda, M. (January 2003). "Oral treponemes in primary root canal infections as detected by nested PCR". International Endodontic Journal. 36 (1): 20–26. doi:10.1046/j.0143-2885.2003.00607.x. ISSN 0143-2885.
  4. ^ Sivapathasundharam, B (July 2022). "Equally credited authors". Journal of Oral and Maxillofacial Pathology. 26 (3): 307–308. doi:10.4103/jomfp.jomfp_30_22. ISSN 0973-029X. PMC 9802513. PMID 36588847.
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  6. ^ Jevon, Phil; Abdelrahman, Ahmed; Pigadas, Nick (September 2020). "Management of odontogenic infections and sepsis: an update". British Dental Journal. 229 (6): 363–370. doi:10.1038/s41415-020-2114-5. ISSN 0007-0610. PMC 7517749. PMID 32978579.
  7. ^ Bansal, Shweta; Garg, Arun; Khurana, Richa; Bansal, Archisha (2020). "Primary orofacial granulomatous involvement of lip and gingiva only: A diagnostic challenge". Journal of Indian Society of Periodontology. 24 (6): 575. doi:10.4103/jisp.jisp_18_20. ISSN 0972-124X. PMC 7781253. PMID 33424177.
  8. ^ "Magnetic resonance imaging of the temporomandibular joint : Normal appearances., Price C, et al, Dentomaxillofac, Radiol, 15(2), 79-85, (1986)". Japanese Journal of Radiological Technology. 43 (6): 730. 1987. doi:10.6009/jjrt.kj00001363134. ISSN 0369-4305.
  9. ^ Hargreaves, Kenneth M.; Cohen, Stephen; Berman, Louis H. (2011), "Preface", Cohen's Pathways of the Pulp, Elsevier, pp. ix, doi:10.1016/b978-0-323-06489-7.00034-5, ISBN 978-0-323-06489-7, retrieved 2025-03-30